Talk:Sodium-glucose transport proteins

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1 Aug 2014- Empagliflozin was approved (Jardiance) by the FDA for use in DMT2.

http://us.boehringer-ingelheim.com/news_events/press_releases/press_release_archive/2014/08-01-14-fda-approves-jardiance-empagliflozin-tablets-reduce-blood-sugar-levels-adults-type-2-diabetes.html — Preceding unsigned comment added by 162.134.56.6 (talk) 17:52, 5 November 2014 (UTC)[reply]

Clarification needed: sodium depletion or rise in potassium?

In "Function" section I read:

sodium can deplete, so Sodium–hydrogen antiporter gets sodium into the cell to begin with.

but SGLT imports in the cell glucose and sodium, thus I see no reason for sodium depletion.

On the other hand, it is stated that Na⁺/K⁺ ATPase exports sodium and imports potassium, thus I think the limiting factor should be rise in potassium concentration.

Someone with sufficient knowledge of this matter can clarify? Fornaeffe (talk) 16:00, 14 October 2024 (UTC)[reply]

I found this section hard to follow, so I rewrote it. Hopefully it is now clearer and more accurate. Also this section is about mechanism, not function, so I renamed it. Boghog (talk) 06:20, 15 October 2024 (UTC)[reply]